Hi. I'm Deep Relief.
I attach to your CB2 receptors (like CBD) to regulate and reduce pain and inflammation, extend the action of endogenous acetylcholine, inhibit pain transmition cells, increase adenosine levels in the brain and stimulate the opioidergic, cholinergic and dopaminergic systems that regulate sensation.
Beta-Caryophyllene: BCP has been shown to attach to the same receptors as CBD (CB2 receptors). This activation may result in a reduction of inflammatory and neuropathic pain. BCP has demonstrating efficacy in enhancing osteoblastic bone mineralisation, as well as decreasing osteo breakdown (adipogenesis and osteoclastogenesis).
Studies indicate that this terpene may assist in pain related to the nervous system, and has demonstrated efficacy as a topical anaesthetic.
BCP has also demonstrated considerable anxiolytic effects in mouse and human models.
Myrcene: Myrcene has demonstrated significant analgesic and anti-inflammatory properties, and has been shown to reduce pain in neuropathic models.
Myrcene is a powerful cannabinoid modulator and enhances both the strength and duration of the pain relieving cannabinoids THC & CBD, allowing them to cross the blood-brain barrier.
Linalool: Linalool has demonstrated effectiveness in reducing several varieties of pain: by reducing acetylcholine at the neuromuscular junction, reducing the excitability of spinal cord cells that transmit pain signals to the brain, and by increasing adenosine levels in the brain and stimulating the opioidergic, cholinergic and dopaminergic systems that regulate sensation.
D-Limonene: D-Limonene has displayed efficacy in reducing neuropathic and chronic muscoluskeletal pain by exerting antihyperalgesic action in the dorsal horn of the spinal cord. D-Limonene is also a significant contributor to the entourage effect.
Camphene: Camphene, as well as enhancing the strength of other terpenes it's with, demonstrates effective anti-inflammatory and analgesic properties on its own.